Serum adenosine deaminase activity and the total antioxidant capacity of plasma in pulmonary tuberculosis and non-tuberculosis pulmonary disease

Annually, more than 8 million people develop tuberculosis (TB), and approximately 1.8 million cases result in death (1). TB has a long incubation period, with the timeline for transition from infection to expression lastingmonths or decades. Therefore, it is not surprising that identification of interspersion transmission events by classical epidemiological tools, like contact tracing, suffer from recall and observer biases (2). The introduction of chemotherapeutic and prophylactic measures had led to a substantial reduction in deaths, which was maintained for decades. Regardless, TB is still responsible for the most human deaths caused by a single infectious agent (3). Pulmonary TB continues to be a major health problem in the Sistan and Balouchistan provinces of Iran, where it is the most prevalent disease. Serum adenosine deaminase activity and the total antioxidant capacity of plasma in pulmonary tuberculosis and non-tuberculosis pulmonary disease